Questions about Diabetes?

Kumar said:
Good words looks bad to you, formal but illogicalwords looks good to you. What to do, just flatter????:(
If you think that the complete nonsense you're posting is "good words", and the patient and clear explanations of Hans and all the others here who are trying to explain the truth to you are "illogical", then you're an even more egotistical fool than I thought.

Rolfe.
 
Mmmmmkay, I'm still missing:

- data on this insulin resistance thing
- data on a salt-blood sugar connection
- what the salt thing has to do with the insulin thing

Got any of that, Kumar?
 
Kumar said:
Insulin can act both as cause & effect. High BG causes more insulin secretion-here High BG is cause & high insulin secretion an effect. Right? Now, as you said "high insulin level due to faulty administering of insulin where sensitizing had been appropriate might speed up the developement of insensitivity", higher insulin level can be a cause to insensitivity of it, due to its excess exposure to cells, excess BG level thus caused by this insestivity so "the effect". Right?

Now say at pre-diabetic stage; due to fault in eating habits (irregular, excessive, occasional & sugar specifics) & due to other modern lifestyle & environment--occasional & excess insulin may be secreted-which may result in speeding up insulin senstivity>>so high BG. The cause here is excess insulin>>if you then firthur increase insulin by medication>>more insenstiveness>>>persistant high BG>>rest you can understand.:(
Kumar, you can repeat your ◊◊◊◊◊◊◊ nonsense all you will. It will remain ◊◊◊◊◊◊◊ nonsense. You don't have a clue, you are incabable of logic. That is tough, but that is how it is.

Hans
 
Kumar said:
BSM, these are bit dynamic/differant aspects. Links may just help in linking but will not in telling exactly. As far as as I could link--these looks bit positive as yet, but you people are specialized in making the most negative, so presenting here for your.....:D

What the heck are you on about?

Use the links you posted to form an argument, you know, a coherent set of thoughts arranged to support a viewpoint.

So, let's try again, supply one piece of evidence that insulin resistance is the result of insulin being given instead of resulting from chronic hyperglycaemia, down-regulation of insulin-receptors and other pathological processes.
 
Kumar said:
TT,

What do you say in consideration of following quote (was also quoted previously in this topic:-

Kumar,

You have repeatedly demonstrated that you just do not have even a basic concept of cellular physiology. As such, there's really no point in getting into sharing my thoughts on sodium-potassium dependent transport mechanisms in the cellular membrane.

Suffice it to say that the connection you are trying to make simply does not exist. This fact has been repeatedly studied and verified in the thousands, if not tens of thousands, of physiologic studies that have been conducted over the past several decades. Furthermore, continuing studies continually reverify our understanding of these mechanisms. For a host of reasons I have a high confidence level that you will not be able to grasp (and I'm not going to waste my energy trying to explain to you), you're continued investigation and instigation is counter productive, and only serves to further confuse you.

In other words, give it up.

-TT
 
TT,

Do you want to say that mentioning on that site is wrong or I am wrongly understanding those mentionings?
 
Zombified said:
Mmmmmkay, I'm still missing:

- data on this insulin resistance thing
- data on a salt-blood sugar connection
- what the salt thing has to do with the insulin thing

Got any of that, Kumar?

BSM: So, let's try again, supply one piece of evidence that insulin resistance is the result of insulin being given instead of resulting from chronic hyperglycaemia, down-regulation of insulin-receptors and other pathological processes.

I have asked the questions & doubts thereon. I already indicated the reasons for doubting the same. Rest you have to clear.

If you have insulin resistance, your muscle, fat, and liver cells do not use insulin properly. The pancreas tries to keep up with the demand for insulin by producing more. Eventually, the pancreas cannot keep up with the body's need for insulin, and excess glucose builds up in the bloodstream. Many people with insulin resistance have high levels of blood glucose and high levels of insulin circulating in their blood at the same time.http://diabetes.niddk.nih.gov/dm/pubs/insulinresistance/

I doubt that higher exposure of insulin results , cells to become its resistant or reduces its sensitiveness. My indications are:Type1 who are also hyperglymic but do not usually become insulin resistant. Anyway, you can explain your theory to it that how cells become insulin resistant? Why Receptors etc. can't be effected by high insulin levels?

Reg: Salt, I have quoted 3 links. Pls tell if those links are wrong or I am wrongly understanding those links?

Mr Hans,

What censored. Nothing was censored.
 
The links are wrong. You couldn't post a coherent argument out of them. Not that you could, or would even try.
 
I have just indicated my doubts. You check these more carefully without any ego..so on. You can check the effects by relating effects by taking sugar, sugar+water, sugar+water+NaCl, sugar+water+NaCl+KCL/Mg.salt/Ca salt.

I may also add one more question:-

How imbalances in digestive pHs & internal pHs can be related to imbalanes in BG(bollod gulucose) levels?
 
Kumar said:
I have just indicated my doubts. You check these more carefully without any ego..so on. *SNIP*
I'd like to clear out a basic misunderstanding here, Kumar: You are NOT a school-master. You are not entitled to just ask questions and demand others investigate for you.

YOU are the pupil here, and it is high time YOU started doing some homework.

Hans
 
Mr.Hans,

You never treated yourself as pupil at homeopathic forums then how can you expect/ suggest others to become pupil here. Moreover, whatever I mentioned here is much more than any type of person, in gross.

I added/adding more question for dynamic discussions:

Q3: How imbalances in digestive pHs & internal pHs can be related to imbalances in BG(bloood gulucose) levels?

Q4: How, weight gain, stay constant, & loose can be linked to various diabetes conditions?

Q5: How daily fluctuations (non-persisting high) & persisting high for long but studden fall, in BG levels can be linked to diabetic conditions & medications?

Q6: Which minerals & trace elements are related to diabetes & its complications?


I think, these questions are sufficient for this topic. Thanks for only contributions in advance.:)
 
Badly Shaved Monkey said:
Supply one piece of evidence that insulin resistance is the result of insulin being given instead of resulting from chronic hyperglycaemia, down-regulation of insulin-receptors and other pathological processes.
 
Kumar for BSM said:
I have asked the questions & doubts thereon. I already indicated the reasons for doubting the same. Rest you have to clear.

I doubt that higher exposure of insulin results , cells to become its resistant or reduces its sensitiveness. My indications are:Type1 who are also hyperglymic but do not usually become insulin resistant. Anyway, you can explain your theory to it that how cells become insulin resistant? Why Receptors etc. can't be effected by high insulin levels?

Reg: Salt, I have quoted 3 links. Pls tell if those links are wrong or I am wrongly understanding those links?

 
Sarah's running again? Say it isn't so! Sarah, I'll try this one more:

Originally posted by Sarah-I
No it does not conflict at all. I am talking about the treatment of chronic cases. Acute cases differ slightly, in that a full case need not always be taken and there is a need to get a remedy in there quickly. Repetition of dose will be more frequent with an acute case too. The one dose that I am talking about refers to the treatment of chronic cases. An acute case will probably need more than one dose of an indicated remedy.


So, what is the basic premise here? You need to see if the remedy works in a chronic case because you don't know whether it will or not? Yet you claim acute cases can be treated, which implies you know the remedy will work. Which is it, do you know remedies work or do you have to tinker with each one. Why is one different from the other? Are the remedies somehow endowed with a sense of urgency and failure-is-not-an-option in the case of acute cases and not so endowed in the case of chronic cases? How on earth is that so?
 
COFFEE BREAK:

Americans Getting Taller, Much Heavier
Americans are getting a little taller and a lot fatter. Adults are roughly an inch taller than they were in the early 1960s, on average, and nearly 25 pounds heavier, the government reported. In 1960-62, the average man weighed 166.3 pounds. By 1999-2002, the average was 191 pounds, according to the report from the National Center for Health Statistics, part of the Centers for Disease Control and Prevention. The report said the average woman's weight rose from 140.2 pounds to 164.3 pounds.

The trends are the same for children, the report said. Average 10-year-olds weighed about 11 pounds more in 1999-2002 than they did 40 years ago. "All the kids who are obese now will become obese adults," a researcher said. "What will happen with the next generation of adults is really scary." Obesity can increase the likelihood of diabetes, high blood pressure and high cholesterol and lead to other health problems.

ADA LINK.

Can you think any possible reason to it?

A Regular Soda a Day Boosts Weight Gain
 
Kumar said:
Can you think any possible reason to it?
Yeah. Too much junk food, not enough exercise. You must have missed ThirdTwin's thread on this subject; he's on that ball already.

Kumar, you're starting to disappoint me again. You never seem to want to answer any questions yourself, and you aren't taking in what people are telling you.

Why do you keep repeating the same questions? Do you think the answers will change if you repeat them often enough?
 
Zombified said:
Why do you keep repeating the same questions? Do you think the answers will change if you repeat them often enough?

"Won't they?"
"No."
"Won't they?"
"No."
"Won't they?"
"NO."
"Won't they?
 
Zombified said:
Yeah. Too much junk food, not enough exercise. You must have missed ThirdTwin's thread on this subject; he's on that ball already.

Kumar, you're starting to disappoint me again. You never seem to want to answer any questions yourself, and you aren't taking in what people are telling you.

Why do you keep repeating the same questions? Do you think the answers will change if you repeat them often enough?

OR,

Modern food/lifestyle/environment/more mental stress>> high stomach acid/imbalance in gastric pH? High gastric acid>>better digestion of many substances>>more absorption>>more body?

More bile>>more fats. Less/more bicarbonate>>>????
 
Kumar said:
NO, Kumar. I'm sure ThirdTwin can give a more nuanced biochemical explanation than I ever will, but to first order, obesity is just a manifestation of conservation of energy. Fat contains around 3,500 kcal/pound. Eat more calories, burn fewer calories, store more fat. It's that simple. Wanna lose it? Toss out the junk food and put on your running shoes.

People today are fatter because calories are cheap and easy to get, back in the day you had to work to get 'em. That is the effect of modern living, more than anything else.
 
Zombified said:
NO, Kumar. I'm sure ThirdTwin can give a more nuanced biochemical explanation than I ever will, but to first order, obesity is just a manifestation of conservation of energy. Fat contains around 3,500 kcal/pound. Eat more calories, burn fewer calories, store more fat. It's that simple. Wanna lose it? Toss out the junk food and put on your running shoes.

People today are fatter because calories are cheap and easy to get, back in the day you had to work to get 'em. That is the effect of modern living, more than anything else.

That can be a tool but I am talking about physiological reason/cause to all imbalanced inputs. It also looks, we are ignoring some "Lactic Acid" consideration in this?
 

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